One complication, a venous tear, occurred related to postconditioning. Postconditioning could be applied all times. Donor age and serum creatinine were higher in the experimental group compared to historical control: 57.7 (20-71) vs. Furthermore, we performed a paired kidney analysis using the contralateral kidney (n=11). Data were compared to a historical control group (n=40). Secondary outcomes were incidence of DGF and renal function at 3 months. Primary outcome was rate of serious adverse events. We performed a pilot study to assess feasibility and safety of ischemic postconditioning in human donation-after-circulatory death kidney transplantation. Ischemic postconditioning may improve outcome after kidney transplantation. Furthermore, living donation remains underused. Potential DBDs and avoid their referral for donation as DCDs before brainĭeath occurs. Trolled DCD programs while simultaneously improve the identiﬁcation of all To increase KT activity, Belgium should further expand con. This could suggest a shift fromĭBDs to DCDs. Increased number of DCD donations and transplantations, the total number ofĭeceased KT did not increase signiﬁcantly.

Solution were independent risk factors for the development of DGF. Machine perfusion), cold ischemic time, and histidine-tryptophan-ketoglutarate In multivariate analysis, cold storage (versus Five-year patient and death-censored graft survival The total KT activity in Belgium were compared with the Netherlands. The evolution of the kidney donor type and the impact of DCDs on Five-year patient/graft survival was assessed using Kaplan–MeierĬurves. Riskįactors for delayed graft function (DGF) were identiﬁed using multivariateĪnalysis. We reviewed the BelgianĭCD kidney transplant (KT) experience since its reintroduction in 2000. Of kidneys donated after circulatory death (DCD). Worldwide shortage of standard brain dead donors (DBD) has revived the use

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